Search results for "Neurotrophin 3"

showing 9 items of 9 documents

Clinical staging and serum cytokines in bipolar patients during euthymia

2017

Aims: Changes in serum cytokines and altered neutrophin concentration have been associated with bipolar disorder (BD). Our aim here was to analyze peripheral blood biomarkers according to the clinical stages of BD. Method: Euthymic BD-I patients were grouped according to their level of functioning in early-stage (n = 25) and late-stage (n = 23), and compared to healthy siblings (n = 23) and genetically unrelated healthy controls (n = 21). Neurotrophin (neurotrophin-3 and BDNF) concentration and biomarkers of inflammation, including cytokines (IL-6, IL-10 and TNF-alpha), leukocytes count and acute phase proteins, were measured. Results: IL-10 concentration was significantly increased in earl…

AdultMaleBipolar DisorderBipolar disorderInflammationNeurotrophinsYoung Adult03 medical and health sciences0302 clinical medicineNeurotrophin 3medicineLeukocytesHumansNerve Growth FactorsBipolar disorderBiological PsychiatryPharmacologyInflammationbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryBrain-Derived Neurotrophic FactorSiblingsAcute-phase proteinMiddle Agedmedicine.diseaseBlood Cell CountInterleukin-10030227 psychiatryPeripheralAffectSerum cytokineClinical stagingCase-Control StudiesImmunologybiology.proteinBiomarker (medicine)CytokinesFemaleTumor necrosis factor alphamedicine.symptombusinessBiomarkers030217 neurology & neurosurgeryAcute-Phase ProteinsNeurotrophin
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Evidence of neurotrophic events due to peritoneal endometriotic lesions

2012

To investigate the neurotrophic properties of endometriosis, as well as the involvement of neurotrophic factors in the development of chronic pelvic pain in patients with endometriosis, we performed a prospective clinical study. The presence of neurotrophins was investigated in the peritoneal fluid (PF) of patients with peritoneal endometriotic lesions or adenomyosis, as well as from women with non-endometriotic adhesions and from women without endometriosis/adenomyosis/adhesions. The PF from patients with peritoneal endometriotic lesions was divided in three groups: asymptomatic endometriosis, minimal pain and severe pain. PF from patients with adenomyosis or with non-endometriotic adhesio…

AdultPathologymedicine.medical_specialtyImmunologyEndometriosisEndometriosisTissue AdhesionsBiochemistryCell LineYoung AdultPelvic painNeurotrophin 3Neurotrophic propertieNeurotrophic factorsGanglia SpinalmedicineNeuritesAnimalsAscitic FluidHumansImmunology and AllergyAdenomyosisProspective StudiesEndometriosiMolecular BiologyBrain-derived neurotrophic factorbiologybusiness.industryPeritoneal fluidPelvic painBrain-Derived Neurotrophic FactorMedicine (all)HematologyMiddle Agedmedicine.diseaseNerve growth factorPain generationbiology.proteinFemaleNeurotrophinmedicine.symptomChronic PainPeritoneumbusinessChickensAdenomyosisNeurotrophin
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Postsynaptic Secretion of BDNF and NT-3 from Hippocampal Neurons Depends on Calcium–Calmodulin Kinase II Signaling and Proceeds via Delayed Fusion Po…

2007

The mammalian neurotrophins (NTs) NGF, BDNF, NT-3, and NT-4 constitute a family of secreted neuronal growth factors. In addition, NTs are implicated in several forms of activity-dependent synaptic plasticity. Although synaptic secretion of NTs has been described, the intracellular signaling cascades that regulate synaptic secretion of NTs are far from being understood. Analysis of NT secretion at the subcellular level is thus required to resolve the role of presynaptic and postsynaptic NT secretion for synaptic plasticity. Here, we transfected cultures of dissociated rat hippocampal neurons with green fluorescent protein-tagged versions of BDNF and NT-3, respectively, and identified NT vesi…

Calcium Channels L-TypeBiologyNeurotransmissionInhibitory postsynaptic potentialHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionExocytosisNeurotrophin 3Postsynaptic potentialCa2+/calmodulin-dependent protein kinaseAnimalsCalcium SignalingNeuronsBrain-Derived Neurotrophic FactorGeneral NeuroscienceRyanodine Receptor Calcium Release ChannelLong-term potentiationArticlesCyclic AMP-Dependent Protein KinasesRatsCell biologynervous systemBiochemistryTrk receptorCalcium-Calmodulin-Dependent Protein KinasesSynapsesSynaptic plasticityThapsigarginCalcium-Calmodulin-Dependent Protein Kinase Type 2Postsynaptic densityThe Journal of Neuroscience
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Spatial shaping of cochlear innervation by temporally regulated neurotrophin expression.

2001

Previous work suggested qualitatively different effects of neurotrophin 3 (NT-3) in cochlear innervation patterning in different null mutants. We now show that all NT-3 null mutants have a similar phenotype and lose all neurons in the basal turn of the cochlea. To understand these longitudinal deficits in neurotrophin mutants, we have compared the development of the deficit in the NT-3 mutant to the spatial–temporal expression patterns of brain-derived neurotrophic factor (BDNF) and NT-3, using lacZ reporters in each gene and with expression of the specific neurotrophin receptors, trkB and trkC. In the NT-3 mutant, almost normal numbers of spiral ganglion neurons form, but fiber outgrowth t…

HeterozygoteCell SurvivalCell CountNeurotrophin-3Tropomyosin receptor kinase BTropomyosin receptor kinase CArticleMiceNeurotrophin 3Neurotrophic factorsGenes ReportermedicineAnimalsReceptor trkBReceptor trkCNeurons AfferentCochleaSpiral ganglionBrain-derived neurotrophic factorAfferent PathwaysbiologyGeneral NeuroscienceBrain-Derived Neurotrophic FactorHomozygoteGene Expression Regulation DevelopmentalImmunohistochemistryMice Mutant StrainsCochleamedicine.anatomical_structurePhenotypenervous systemAnimals NewbornLac OperonMutationbiology.proteinSpiral GanglionNeuroscienceNeurotrophin
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Mesenchymal Stem Cells Improve Motor Functions and Decrease Neurodegeneration in Ataxic Mice

2014

The main objective of this work is to demonstrate the feasibility of using bone marrow-derived stem cells in treating a neurodegenerative disorder such as Friedreich's ataxia. In this disease, the dorsal root ganglia of the spinal cord are the first to degenerate. Two groups of mice were injected intrathecally with mesenchymal stem cells isolated from either wild-type or Fxntm1Mkn/Tg(FXN)YG8Pook (YG8) mice. As a result, both groups presented improved motor skills compared to nontreated mice. Also, frataxin expression was increased in the dorsal root ganglia of the treated groups, along with lower expression of the apoptotic markers analyzed. Furthermore, the injected stem cells expressed th…

Malemedicine.medical_specialtyAtaxiaCellular differentiationGene ExpressionBone Marrow CellsMice TransgenicMotor ActivityMesenchymal Stem Cell TransplantationTransplantation AutologousMiceGlutathione Peroxidase GPX1Neurotrophin 3Internal medicineGanglia SpinalIron-Binding ProteinsDrug DiscoverymedicineGeneticsAnimalsTransplantation HomologousNerve Growth FactorsMolecular BiologyInjections SpinalPharmacologyGlutathione PeroxidasebiologyBrain-Derived Neurotrophic FactorMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAnatomySpinal cordCatalaseDisease Models AnimalEndocrinologymedicine.anatomical_structureFriedreich AtaxiaFrataxinbiology.proteinMolecular MedicineOriginal ArticleFemaleBone marrowmedicine.symptomStem cellAdult stem cell
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Updated overview on interplay between physical exercise, neurotrophins, and cognitive function in humans

2019

Highlights • Physical exercise maintains or improves cognitive functions, and neurotrophin receptor signaling may play an important role. • A positive relationship exists between physical exercise and circulating brain-derived neurotrophic factor levels. • Less clear evidence has been found for a relationship between physical exercise and other neurotrophin levels, such as nerve growth factor, neurotrophin-3, and neurotrophin-4. • The postexercise variation of brain-derived neurotrophic factor might be associated with improvement of neurocognitive functioning. • Physical exercise may be an inexpensive and safe strategy for boosting brain-derived neurotrophic factor release, thus preserving …

Nerve Tissue ProteinsPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseReceptors Nerve Growth Factorneurotrophins03 medical and health sciencesCognition0302 clinical medicineNeurotrophin 3Neurotrophic factorsphysical exerciseRegular PaperHumansMedicineAerobic exerciseOrthopedics and Sports MedicineNerve Growth Factors030212 general & internal medicineExercisephysical exercise neurotrophins cognitive functioncognitive functionSportbiologybusiness.industryBrain-Derived Neurotrophic FactorCognition030229 sport sciencesNerve growth factorSynaptic plasticitybiology.proteinbusinessNeuroscienceNeurocognitiveNeurotrophin
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Endothelial NT-3 Delivered by Vasculature and CSF Promotes Quiescence of Subependymal Neural Stem Cells through Nitric Oxide Induction

2014

SummaryInteractions of adult neural stem cells (NSCs) with supportive vasculature appear critical for their maintenance and function, although the molecular details are still under investigation. Neurotrophin (NT)-3 belongs to the NT family of trophic factors, best known for their effects in promoting neuronal survival. Here we show that NT-3 produced and secreted by endothelial cells of brain and choroid plexus capillaries is required for the quiescence and long-term maintenance of NSCs in the mouse subependymal niche. Uptake of NT-3 from irrigating vasculature and cerebrospinal fluid (CSF) induces the rapid phosphorylation of endothelial nitric oxide (NO) synthase present in the NSCs, lea…

Nitric Oxide Synthase Type IIICell SurvivalNeuroscience(all)BiologyNitric OxideNitric oxidechemistry.chemical_compoundMiceCerebrospinal fluidNeural Stem CellsNeurotrophin 3Subependymal zoneAnimalsCells CulturedCell ProliferationNeuronsGeneral NeuroscienceEndothelial CellsCell DifferentiationNeural stem cellCell biologynervous systemchemistrybiology.proteinPhosphorylationChoroid plexusStem cellNeuroscienceNeurotrophinNeuron
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Role of interleukin-6 and soluble IL-6 receptor in region-specific induction of astrocytic differentiation and neurotrophin expression.

1999

Increasing evidence supports an essential role for interleukin-6 (IL-6) in the development, differentiation, as well as de- and re-generation of neurons in the central nervous system (CNS). Both IL-6 and its specific receptor (IL-6R) are expressed on neurons and glial cells including astrocytes. In this study, we have analyzed the responses of primary rat astrocytes of various brain regions to IL-6 with respect to morphological changes and neurotrophin expression. Since IL-6 alone failed to initiate effects on astrocytes, we have examined whether the soluble IL-6R (sIL-6R) can modulate the responsiveness of to IL-6 in these cells. For this purpose, we used a highly active fusion protein of …

Recombinant Fusion ProteinsCentral nervous systemHippocampusNeurotrophin-3HippocampusImmunoenzyme TechniquesRats Sprague-DawleyCellular and Molecular NeuroscienceNeurotrophin 3medicineAnimalsHumansNerve Growth FactorsCells CulturedCerebral CortexbiologyInterleukin-6Reverse Transcriptase Polymerase Chain ReactionCell DifferentiationImmunohistochemistryReceptors Interleukin-6RatsBlotting Southernmedicine.anatomical_structureNerve growth factornervous systemNeurologyAnimals NewbornCerebral cortexAstrocytesbiology.proteinNeurogliaNeuroscienceAstrocyteNeurotrophinGlia
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Expression of neurotrophins, GDNF, and their receptors in rat thyroid tissue

1999

Levels of mRNA for neurotrophins (brain-derived neurotrophic factor, BDNF; neurotrophin 3, NT-3; neurotrophin 4, NT-4) and their receptors (trkA, trkB, trkC) and for glial cell line-derived neurotrophic factor (GDNF) and its receptors (ret, GDNFR-alpha) were measured in rat thyroid tissue by ribonuclease protection assays. In thyroid tissue the NT-3 mRNA level was threefold lower and the NT-4 mRNA level sixfold higher than those detected in adult rat hippocampus, while BDNF mRNA was undetectable. Very low levels of mRNA for truncated trkB and trkC receptors and no catalytic trkA, trkB or trkC were found. In conclusion NT-3 and NT-4, but not the corresponding functional receptors, are expres…

endocrine systemmedicine.medical_specialtyGlial Cell Line-Derived Neurotrophic Factor ReceptorsHistologyendocrine system diseasesThyroid GlandGene ExpressionNerve Tissue ProteinsReceptors Nerve Growth FactorNeurotrophin-3Tropomyosin receptor kinase AFollicular cellPathology and Forensic MedicineNeurotrophin 3Proto-Oncogene ProteinsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsDrosophila ProteinsHumansLow-affinity nerve growth factor receptorReceptor trkCGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsRNA MessengerReceptor trkAReceptor Ciliary Neurotrophic FactorbiologyBrain-Derived Neurotrophic FactorProto-Oncogene Proteins c-retReceptor Protein-Tyrosine KinasesCell BiologyRatsCell biologyEndocrinologynervous systemProto-Oncogene Proteins c-retbiology.proteinGDNF family of ligandsNeurotrophinCell and Tissue Research
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